D. The decline in clinical sequelae of genital Chlamydia trachomatis infection supports current control strategies. kb plasmid [ 113, 114], and the ability of the pathogen to alter the proteome profile of the host cells [ 115, 116]. pneumoniae strains may have originated in non-human animals, which have gradually adapted to human hosts through progressive loss in certain genes and plasmids, and ultimately sidestepping the requirement for animal reservoirs [ 127].

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